Blood vessels in the body will be destroyed by high blood pressure, and the pressure of the blood to push the human artery can strain or tear the cell wall of the artery. The injured arteries can repair itself, but when the damage is too large or too frequent, the repair process will produce a bulge of the fat, cholesterol, and dead cells (also known as fat plaques). For people with elevated blood cholesterol levels, especially those with low density lipoprotein cholesterol, the risk of the fatty plaques is the most dangerous. High blood pressure will speed up the process, and the fat spot will continue to grow until it is long enough to block the flow of blood. This process is the process of atherosclerosis, which may occur from childhood and has been sustained for several years 1-. Do the same in the hypertension, atherosclerosis caused enough damage to cause obvious symptoms before is soundless and stirless. In nearly 1/5 of patients with atherosclerosis, fatal heart attack is the only symptom.
In addition to accelerate artery plugging, hypertension can accelerate atherosclerosis (hardening of the arteries) process. Arterial blood is thick and stiff, and it will cause the arteries to narrow and make the arteries lose their elasticity, so that the process of atherosclerosis is accelerated. The results are less and less blood in the blood vessels through the blood vessels.
Hypertension accelerates the process of atherosclerosis by causing tissue damage to the arterial wall. When blood pressure is too high, excessive blood pressure can be struck on the fragile artery wall, which can cause damage to blood vessels. Other arteries are also hurt by the harmful chemical substances in cigarettes and the low density lipoprotein cholesterol (chemical change). Foods rich in antioxidant vitamins (especially vitamin A, vitamin C, and vitamin E) can reduce the amount of oxidized LDL cholesterol in the blood, which is contained in foods such as citrus fruits, leafy vegetables, Cereals, and low fat dairy products.
Attract impaired movement in the event a called white Xirun mononuclear cells, they are inflammatory attack the blood of bacteria and other foreign substances of white blood cells, these mononuclear cell attachment in the damaged area to complete their work, and eventually drilling in the inner lining of the arteries of the internal.
Once in the human arterial union internal, mononuclear cells will into another cell, called macrophages, macrophages in vivo play or doffer. Garbage collection. The role of macrophages in the phagocytosis of dead cells, so that they no longer block blood flow channel, but also to absorb low density lipoprotein cholesterol. Dolphin alliance of macrophages are not and animals by blood circulation, and so they will stay forever in the Ministry when low density lipoprotein in the blood of the finding in human atherosclerotic lesions of the pathway in macrophages will its absorption and expansion become foam cells. If low density lipoprotein cholesterol levels are elevated in the blood, LDL may be more prone to impaired blood vessel regions. So as to promote the formation of foam cells in the formation of macrophages. When the foam cells gathered together, a fat (i.e. fatty plaques) formed in animal dolphin house.
As the foam cells continue to grow, they begin to crush the arteries, making the injured area expand and prevent blood flow. This additional damage to the artery to attract other blood platelets — Tian Yu one. Platelets remain in the damaged site to release a chemical, which promotes the reverse growth of the fragile arteries. Smooth muscle in the arterial wall can also react to the chemical and begin to reverse its growth. By the accumulation of stem cells and the abnormal growth of tissue, the arteries will become narrower and eventually become completely blocked.
In the damaged site, the clot aggregates the calcium, fibrous tissue, and other debris in the blood that increases the risk of arterial blockage. Blood clots may be from the artery (especially in additional pressure exerted by hypertension) can clog arteries and cause stroke or myocardial infarction.
Coronary artery disease occurs when the arteries that supply blood to the heart are narrowed (usually due to the accumulation of plaques). Ischemic heart disease (ischemia) is the result of the occurrence of coronary artery atherosclerosis, or coronary artery spasm (due to the involuntary contraction of the arterial wall muscles) and the results of contraction. Sometimes, such a narrow narrow will not cause any discomfort, this situation is called asymptomatic ischemia. However, most people in the tissue ischemia and hypoxia will occur angina (chest pain). The position of angina occurs in the chest, but may spread to the neck, shoulders, arms, or jaw. Angina attack does not cause permanent damage, but the attack of angina pectoris is an important warning signal.
The condition of the arterial obstruction can occur in any part of the body. The leg artery coarctation can cause pain or cramps when you walk, this is called intermittent claudication. Like angina, the pain disappears after a break. However, severe untreated leg artery occlusion may eventually result in tissue necrosis of the toe (and leg). This is called gangrene, especially in some diabetic patients.
The arteries that supply blood to the arteries are narrowed, which can cause stomach and stomach cramps. This is abdominal pain, usually in the meal after the pain. The blood vessel that leads to the intestinal tract is completely blocked can cause blood in the stool, and eventually the blockage of the intestinal tract may be necrotic. If the bowel perforation occurs, the bacteria can enter the abdomen, causing abdominal infection (Fu Moyan) or death.
There is another danger, that is, that some of the blocks may fall off and into the blood. If such obstruction is attached to blood vessels and blocks the flow of blood, the tissue of the distal end may begin to necrosis by ischemia. If this occurs in the heart, it is likely to cause myocardial infarction $if the brain occurs in the brain, it will have a stroke.
If blood pressure rises sharply, the risk will be even greater. Extremely high blood pressure can cause the fragile blood vessels to rupture, and the blood flow out of the blood vessel can be seriously damaged by the blood. In the case of the brain, a fatal stroke occurs. The largest artery in the human body, the aorta, is swollen with a high blood pressure. This swelling is the aneurysm, which may rupture. However, the potentially fatal effects of these are caused by a rapid rise in blood pressure that are generally associated with symptoms such as headache, dizziness, nasal bleeding, blurred vision, occasional chest pain or abdominal pain.